tlr2 specific antibody Search Results


primary antibody specific for tlr2 mbs850908  (MyBiosource Biotechnology)
90
MyBiosource Biotechnology primary antibody specific for tlr2 mbs850908
Primary Antibody Specific For Tlr2 Mbs850908, supplied by MyBiosource Biotechnology, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/primary antibody specific for tlr2 mbs850908/product/MyBiosource Biotechnology
Average 90 stars, based on 1 article reviews
primary antibody specific for tlr2 mbs850908 - by Bioz Stars, 2026-03
90/100 stars
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antibodies against tlr2, nf-κb p65, p-nf-κb p65  (Wanleibio)
90
Wanleibio antibodies against tlr2, nf-κb p65, p-nf-κb p65
<t>TLR2/TLR4/NF-κB</t> p65 pathway was involved in the macrophage M1 polarization induced by LPS pre-sEVs. a After macrophages were co-cultured with LPS pre-sEVs, RT-PCR results showed that LPS pre-sEVs could significantly increase the expression of CD80, M1 marker, and inflammatory cytokines TNF-α and IL-6 on macrophages compared with untreated group, whereas the expression of CD206 and CD163, two M2 markers, and anti-inflammatory cytokine TGF-β was decreased. b , c The levels of <t>TLR2,</t> TLR4, and p-NF-κB p65 were significantly up-regulated after LPS pre-sEV treatment compared with the control group. * p < 0.05. ** p < 0.01. *** p < 0.001. **** p < 0.0001. ns, not significant.
Antibodies Against Tlr2, Nf κb P65, P Nf κb P65, supplied by Wanleibio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/antibodies against tlr2, nf-κb p65, p-nf-κb p65/product/Wanleibio
Average 90 stars, based on 1 article reviews
antibodies against tlr2, nf-κb p65, p-nf-κb p65 - by Bioz Stars, 2026-03
90/100 stars
  Buy from Supplier

Image Search Results


TLR2/TLR4/NF-κB p65 pathway was involved in the macrophage M1 polarization induced by LPS pre-sEVs. a After macrophages were co-cultured with LPS pre-sEVs, RT-PCR results showed that LPS pre-sEVs could significantly increase the expression of CD80, M1 marker, and inflammatory cytokines TNF-α and IL-6 on macrophages compared with untreated group, whereas the expression of CD206 and CD163, two M2 markers, and anti-inflammatory cytokine TGF-β was decreased. b , c The levels of TLR2, TLR4, and p-NF-κB p65 were significantly up-regulated after LPS pre-sEV treatment compared with the control group. * p < 0.05. ** p < 0.01. *** p < 0.001. **** p < 0.0001. ns, not significant.

Journal: Inflammation

Article Title: Small Extracellular Vesicles from Periodontal Ligament Stem Cells Primed by Lipopolysaccharide Regulate Macrophage M1 Polarization via miR-433-3p Targeting TLR2/TLR4/NF-κB

doi: 10.1007/s10753-023-01845-y

Figure Lengend Snippet: TLR2/TLR4/NF-κB p65 pathway was involved in the macrophage M1 polarization induced by LPS pre-sEVs. a After macrophages were co-cultured with LPS pre-sEVs, RT-PCR results showed that LPS pre-sEVs could significantly increase the expression of CD80, M1 marker, and inflammatory cytokines TNF-α and IL-6 on macrophages compared with untreated group, whereas the expression of CD206 and CD163, two M2 markers, and anti-inflammatory cytokine TGF-β was decreased. b , c The levels of TLR2, TLR4, and p-NF-κB p65 were significantly up-regulated after LPS pre-sEV treatment compared with the control group. * p < 0.05. ** p < 0.01. *** p < 0.001. **** p < 0.0001. ns, not significant.

Article Snippet: The protein samples were subjected to sodium dodecyl sulfate polyacrylamide gel electrophoresis and incubated with desired antibodies against toll-like receptor (TLR) 4 (Santa Cruz, USA), TLR2, NF-κB p65, p-NF-κB p65 (Wanleibio, China), and GAPDH overnight at 4 °C.

Techniques: Cell Culture, Reverse Transcription Polymerase Chain Reaction, Expressing, Marker, Control

MiR-433-3p is involved, at least in part, in M1 macrophage polarization by LPS pre-sEVs. a Transfection of miR-433-3p inhibitor could significantly reduce the expression of miR-433-3p. b RT-PCR assays demonstrated that the pro-inflammatory TNF-α was significantly decreased after application of miR-433-3p inhibitor. c , d MiR-433-3p inhibitor could inhibit the expression of TLR2 and TLR4 and the phosphorylation of NF-κB p65. *** p < 0.001.**** p < 0.0001.

Journal: Inflammation

Article Title: Small Extracellular Vesicles from Periodontal Ligament Stem Cells Primed by Lipopolysaccharide Regulate Macrophage M1 Polarization via miR-433-3p Targeting TLR2/TLR4/NF-κB

doi: 10.1007/s10753-023-01845-y

Figure Lengend Snippet: MiR-433-3p is involved, at least in part, in M1 macrophage polarization by LPS pre-sEVs. a Transfection of miR-433-3p inhibitor could significantly reduce the expression of miR-433-3p. b RT-PCR assays demonstrated that the pro-inflammatory TNF-α was significantly decreased after application of miR-433-3p inhibitor. c , d MiR-433-3p inhibitor could inhibit the expression of TLR2 and TLR4 and the phosphorylation of NF-κB p65. *** p < 0.001.**** p < 0.0001.

Article Snippet: The protein samples were subjected to sodium dodecyl sulfate polyacrylamide gel electrophoresis and incubated with desired antibodies against toll-like receptor (TLR) 4 (Santa Cruz, USA), TLR2, NF-κB p65, p-NF-κB p65 (Wanleibio, China), and GAPDH overnight at 4 °C.

Techniques: Transfection, Expressing, Reverse Transcription Polymerase Chain Reaction, Phospho-proteomics